Abstract
Human rhinoviruses (HRV) are the most common cause of asthma exacerbations. Reduced production of interferons (IFNs) by bronchial epithelial cells (BECs) may increase susceptibility to HRV infection and illness severity. Therefore, supplementing this attenuated innate immune response is a plausible therapeutic approach for HRV-induced asthma exacerbations. Thus, we investigated the effects of exogenous IFNs on HRV replication in BECs.
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