Abstract

P. Hasbak, C. Lundby, N.V. Olsen, S. Schifter, and I.L. Kanstrup Department of Clinical Physiology and Nuclear Medicine, University Hospital of Herlev, Herlev; Department of Clinical Physiology and Nuclear Medicine, University Hospital of Glostrup, Glostrup; Department of Clinical Experimental Research, University Hospital of Glostrup, Glostrup; Department of Pharmacology, Panum Institute, University of Copenhagen, Denmark

Highlights

  • To date calcitonin gene-related peptide (CGRP) is the most potent vasodilatatory peptide described

  • Hypoxia, lactic acid (LA) and sympathetic activity affect the release of CGRP

  • We studied how CGRP levels would be influenced by maximal exercise in humans during normoxia, acute and prolonged hypoxia

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Summary

Introduction

To date calcitonin gene-related peptide (CGRP) is the most potent vasodilatatory peptide described. EXERCISE-INDUCED CALCITONIN GENE-RELATED PEPTIDE (CGRP) INCREASE DURING NORMOXIA AND HYPOXIA I.L. Kanstrupa aDepartment of Clinical Physiology and Nuclear Medicine, University Hospital of Herlev, Herlev; bDepartment of Clinical Physiology and Nuclear Medicine, University Hospital of Glostrup, Glostrup; cDepartment of Clinical Experimental Research, University Hospital of Glostrup, Glostrup; dDepartment of Pharmacology, Panum Institute, University of Copenhagen, Denmark

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