Abstract
Background: Type 2 diabetes (T2D) is often considered to result from impaired insulin action or secretion. It is well established that exercise can improve glycemic control by increasing insulin sensitivity. However, a much less recognized characteristic of T2D is that, despite the presence of hyperglycemia, counter-regulatory hormones, such as glucagon, often fail to be suppressed postprandially. Furthermore, very little is known about the effect of exercise on subsequent glucagon responses.
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