Abstract
Although Vitamin B12 deficiency affected the central nervous system, normal findings were obtained in C-VEMP testing. In vitamin B12 deficiency, the saccular, inferior vestibular nerve, lateral vestibular nucleus and medial vestibular pathway are not affected.
Highlights
In vitamin B12 deficiency, the function of nerve cells can be disrupted
Vitamin B12 deficiency affected the central nervous system, normal findings were obtained in cervical-vestibular-evoked myogenic potential (C-Vestibular Evoked Myogenic Potentials (VEMPs)) testing
The purpose of this study is to evaluate the functions of the medialvestibular, inferiorvestibular nerve, lateralvestibular nucleus path on adults with vitamin B12 deficiency
Summary
In vitamin B12 deficiency, the function of nerve cells can be disrupted. Vitamin B12 is responsible for balancing neural activity. B12 is an essential cofactor for the methylation of proteins of myelin and cells membrane. In vitamin B12 deficiency; related to demyelization, axonal degeneration and death of neurons, neurological problems emerge [1,2,3,4]. Vitamin B12 deficiency triggered hyperhomocysteinemia has been shown to be significantly associated with neurodegenerative diseases, cognitive impairments, auditory dysfunction, and neuropsychiatric symptoms [5]. Vitamin B12 deficiency has been shown to affect the peripheral vestibular system in some studies [6,7]. The studies on which both peripheral and the central system are not be found in the literature
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