Abstract
Acne is a chronic disease of the pilosebaceous unit. Much attention is paid to the pathogenesis and optimization of acne therapy. Evidence is emerging about the role of PPARγ receptors in maintaining inflammation and increasing sebum production, as well as an indirect role in the development of pruritus, which may allow for a correlation between the severity of inflammation in acne, the prevalence of pruritus, and the influence of PPARγ receptor expression on these processes.
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