Abstract
The sequence of benign peptic ulceration followed by gastritis which, on healing, produces hypertrophic gastric rugae and later a prolapse of gastric mucosa into the duodenal bulb has gained credence as the possible etiological mechanism in this presented case. It is possible that this mechanism plays a more general role in the causation of prolapse. An attempt is made to explain the findings of two other observers who have recently reported on prolapse of the gastric mucosa on the same basis.
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