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Abstract
The effect of acute ethanol (EtOH) exposure on 5-HT 3 receptor-mediated ion current was examined in whole-cell patch-clamp recordings from NCB-20 neuroblastoma cells. The physiologic and pharmacologic properties of 5-HT-activated ion current in NCB-20 cells indicated that it was mediated by 5-HT 3 receptors. EtOH potentiated 5-HT 3 receptor-mediated current in a concentration-dependent manner at concentrations (25–100 mM) which are achieved during EtOH intoxication in vivo.
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