Abstract
High levels of androstenone and skatole in fat tissues are considered the primary causes of boar taint, an unpleasant odour and flavour of the meat from non-castrated male pigs. The aim of this article is to review our current knowledge of the biology and genetic control of the accumulation of androstenone and skatole in fat tissue. Two QTL mapping studies have shown the complexity of the genetic control of these traits. During the last ten years, several authors have taken a more physiological approach to investigate the involvement of genes controlling the metabolism of androstenone and skatole. Although some authors have claimed the identification of candidate genes, it is more appropriate to talk about target genes. This suggests that genes affecting androstenone and skatole levels will have to be sought for among specific or non-specific transcription factors interacting with these target genes.
Highlights
It has been drawn to the attention of the authors that in Figure 1 the common intermediate products of the transformation of progesterone and 5,16 androstadien-3bol into androstenone is 4,16-androstadien-3-ene
The list of the final products of androstenone sulfonation has been completed. This corrigendum published in issue 5, 2008 is freely available in electronic form on the web site of GSE
*Corresponding author: annie.robic@toulouse.inra.fr Article published by EDP Sciences
Summary
It has been drawn to the attention of the authors that in Figure 1 the common intermediate products of the transformation of progesterone and 5,16 androstadien-3bol into androstenone is 4,16-androstadien-3-ene (and not 4,16 androstadien-3-one).
Sign-in/Register to view highlights & summary Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
