Errata

Abstract

The Journal of PhysiologyVolume 564, Issue 3 p. 953-953 Free Access Errata This article corrects the following: Two developmental switches in GABAergic signalling: the K+–Cl− cotransporter KCC2 and carbonic anhydrase CAVII Claudio Rivera, Juha Voipio, Kai Kaila, Volume 562Issue 1The Journal of Physiology pages: 27-36 First Published online: December 22, 2004 Calcium transients in developing mouse skeletal muscle fibres Joana Capote, Pura Bolaños, Ralph Peter Schuhmeier, Werner Melzer, Carlo Caputo, Volume 564Issue 2The Journal of Physiology pages: 451-464 First Published online: April 6, 2005 Excitatory purinergic neurotransmission in smooth muscle of guniea-pig taenia caeci Yong Zhang, William G. Paterson, Volume 563Issue 3The Journal of Physiology pages: 855-865 First Published online: March 8, 2005 First published: 21 April 2005 https://doi.org/10.1113/jphysiol.2005.564001Citations: 1AboutSectionsPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onFacebookTwitterLinkedInRedditWechat The authors of Rivera et al. (2005) would like to publish the following clarification: in our review, we emphasized the fact that using Cs+ as an internal cation in whole-cell recordings results in a profound dysfunction of the neuronal K+–Cl− cotransporter, KCC2. This is because Cs+ is a very poor substrate of this ion transporter, with a maximum transport rate (Vmax) for Cs+ that is < 20% of the value for K+ (Williams & Payne, 2004). On page 29, we stated that ‘Unfortunately, the kinetic data in the only published model of neuronal K+–Cl− cotransport in neurones (Staley & Proctor, 1999) were obtained using Cs+ in whole-cell experiments – and hence, in the absence of functional KCC2!’ Since the publication of our review, it has been brought to our attention that both K+ and Cs+ were used by the authors, with essentially similar results. This is true and, if anything, it strengthens our concerns about the validity of the above modelling study which yielded a value of Vmax for Cs+ that is higher than for K+ (about 7 mmol l−1 s−1 for Cs+ and about 5–6 mmol l−1 s−1 for K+). The authors of Zhang & Paterson, (2005) would like to correct an error on page 858. In line 7 of the first paragraph, a value of 361 ± 83 ms is given for the control. This value should have read 661 ± 83 ms. In addition, in the title ‘guniea-pig’ should be ‘guinea-pig’. The authors of Capote et al. (2005) would like to correct the affiliation of Carlo Caputo; the affiliation should be the Instituto Venezolano de Investigaciones Cientificas IVIC. References Capote J, Bolanos P, Schuhmeier RP, Melzer W & Caputo C (2005). Calcium transients in developing mouse skeletal muscle fibres. J Physiol 564, 451– 464. Rivera C, Voipio J & Kaila K (2005). Two developmental switches in GABAergic signalling: the K+–Cl− cotransporter KCC2 and carbonic anhydrase CAVII. J Physiol 562, 27– 36.DOI: 10.1113/jphysiol.2004.077495 Staley KJ & Proctor WR (1999). Modulation of mammalian dendritic GABAA receptor function by the kinetics of Cl− and HCO3− transport. J Physiol 519, 693– 712.DOI: 10.1111/j.1469-7793.1999.0693n.x Williams JR & Payne JA (2004). Cation transport by the neuronal K+–Cl− cotransporter KCC2: thermodynamics and kinetics of alternate transport modes. Am J Physiol Cell Physiol 287, C919– C931. Zhang Y & Paterson W (2005). Excitatory purinergic neurotransmission in smooth muscle of guniea-pig taenia caeci. J Physiol 563, 855– 865.DOI: 10.1113/jphysiol.2004.077636 Citing Literature Volume564, Issue3May 2005Pages 953-953 ReferencesRelatedInformation