Abstract

Compression stenosis of the celiac trunk or Dunbar syndrome, a clinical and anatomical syndrome characterized by chronic abdominal pain, is a congenital pathology based on compression of the celiac trunk by the median arcuate ligament of the diaphragm. The narrowing of the lumen of the celiac trunk entails hemodynamic disturbances in the artery itself and branches extending from it, which leads to a shortage of blood supply to the organs of the digestive system. Inadequate supply of arterial blood can lead to dystrophic and ischemic tissue damage. The mucous membrane and submucosal layer of the wall of the stomach and intestines are most susceptible to oxygen starvation. The article discusses the features of ischemic gastric and duodenal ulcers with clinical examples in children with Dunbar syndrome. It also describes the compensatory capabilities of the body to minimize the effects of extravasal compression of the celiac artery and methods for their visualization.

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