Abstract
An association between long chain 3-hydroxyacyl-CoA dehydrogenase (LCHAD) deficiency and acute fatty liver of pregnancy (AFLP) has been demonstrated in many reports. The most common variant in clinical observations is the 1528G>C. Our goal was to evaluate the different fetal/neonatal mitochondrial fatty acid oxidation (FAO) genetic variants associated with AFLP in the literature.
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