Abstract
Hypoxic preconditioning and pretreatment with pentoxifylline, α-tocopherol, or their combination promotes stabilization of Po2 in rabbit kidney cortex in the early reperfusion period (in experiments with α-tocopherol at a significantly lower level), while after 30-min reperfusion, the protective effect of α-tocopherol (especially in combination with pentoxifylline) markedly surpasses that of pentoxifylline and hypoxic preconditioning. α-Tocopherol sharply inhibits LPO in the reperfusion period, while pentoxifylline exhibits only weak antioxidant activity and hypoxic preconditioning was ineffective.
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