Abstract
A study was undertaken to evaluate oxidative stress and oxidative defense mechanisms in 14 cases of Eales disease with retinal periphlebitis and 10 controls. Alterations in activity of the enzymes sueeroxide dismutase and catalase in platelets and the lipid peroxidation product malondialdehyde were estimated biochemically. Enhanced oxidative stress (significantly increased malondialdehyde levels and decreased superoxide dismutase enzyme activity) was detected. Results suggest involvement of free radicals in the pathogenesis of Eales disease.
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