Abstract
It has long been recognised that prolonged exposure to high inspired oxygen concentrations can produce pulmonary damage. Exposure to 100% O2 at 1 atm. of pressure is lethal to most mammalian species after a period of days to weeks (1). While the potential dangers of hyperoxia on the lung are generally recognised, administration of above ambient O2 tensions remains a necessity in the treatment of severe hypoxemia caused by respiratory failure or acute lung injury.
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