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Abstract
Of the candidate genes thought to be logical for an individual's hypertension susceptibility, endothelial nitric oxide (NO) synthase gene (protein: eNOS and gene: eNOS) ranks high. Evidence is mounting suggesting that enhanced production or bioavailability of the enzyme eNOS that synthesizes NO in endothelial cells may lead to the constitutive release of NO, which exerts vasoprotective effects in blood pressure (BP) regulation.
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