Abstract
The endothelium has a central role in the regulation of vascular tone, thrombosis, and hemostasis. Endothelial damage incurred during reperfusion injury may limit the restoration of blood flow to potentially viable ischemic myocardium. Although endothelial dysfunction after reperfusion has been demonstrated in animal models, 1, 2 patient data are limited. This study investigates endothelial dysfunction after reperfusion in acute myocardial infarction (MI) patients. Endothelial injury releases factor VIII von Willebrand factor antigen (fVIIIvWFAg), which can be measured in plasma and serves as a marker of endothelial
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