Abstract
Rheology has profound effects on the rate, structure, and modulation of primary hemostasis. Many of these effects can be studied via real-time, epifluorescence videomicroscopy of platelet adhesion-aggregation to a site of injury to an endothelial cell monolayer exposed to flowing blood. In particular, with the model described, endothelial cells can be shown to be a significant modulator of platelet function at an injury site.
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