Abstract
Background: Retinoic acid (RA) regulates many biologic processes, including differentiation, apoptosis, as well as cell survival. Our laboratory has previously shown that RA mediates the progesterone-dependent induction of 17β-hydroxysteroid dehydrogenase type 2, which catalyzes the conversion of estradiol to estrone, in endometrium but not in endometriosis, because of a defect in endometriotic stromal cells. This defect may involve both the uptake and metabolism of RA.
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