Abstract
Whereas participation of vascular endothelial growth factor (VEGF) in angiogenesis and vasculogenesis during development is well-known [1,2], the role of VEGF in heart and vascular functions, particularly in cardiovascular regulation is relatively lacking. Based on echocardiographic observations in fetal liver kinase-1 (Flk-1) deficient mice, our laboratory demonstrated recently [3] that VEGF sustains myocardial performance by acting on Flk-1, also known as kinase insert domain receptor (KDR), in the heart.
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