Abstract
Objective: There is currently no known comprehensive regulatory system which might explain such diverse events as inhibition of capacitation in semen, and suppression of sperm motility within the oviductal isthmus, and timed capacitation of sperm in the female reproductive tract. Arachidonylethanolamide (AEA), also known as anandamide, is an endogenous ligand for cannabinoid (marijuana-like) receptors. We have previously shown that AEA can block acrosome reactions (AR) and enhance sperm motility in sea urchins. AEA is also produced in the rodent testis and uterus. Is AEA present in human reproductive tract fluids? Does AEA or its analogs alter sperm function in the human?
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