Abstract
Capillary clearance and end-vessel reactivity to pressor substances may be factors that could modify wound healing and the response to infection in diabetes mellitus. Angiotensin and saline were injected intradermally in control, diabetic, and atherosclerotic patients to evaluate this hypothesis. Capillary clearance of saline and end-vessel reactivity to angiotensin were both prolonged in the diabetic patient when compared with the control and atherosclerotic patients. Reduced end-vessel perfusion and delayed capillary clearance could combine with the metabolic abnormalities of diabetes mellitus to facilitate the development of infection and to interfere with wound healing.
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