Abstract
The kinetics and diffusion mechanism of surface-injected oxygen interstitials (Oi) in TiO2(1 1 0) were investigated using a microkinetics model in order to determine the effects of different conditions upon to the Oi reaction-diffusion network. Extended defects, whether incipient or pre-existing, may act as a source or sink for Oi. The present work uses a quantitative microkinetic model to describe this diffusion and reaction network based upon isotopic gas-solid exchange experiments. Pertinent outputs from the model determined that the activation barrier for surface injection of Oi is 2.4 eV, while dissolution of Oi from extended defect is 3.3 eV. The interstitial sequestration mechanism through extended defects leads to kinetic coupling effects that other sequestration mechanisms would not typically induce.
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