Abstract
Electrophysiological estimations of the number and size of motor unit action potentials (MUAP) have been made in the extensor digitorum brevis muscle of 9 patients with Duchenne muscular dystrophy and 9 matched control subjects. A critical analysis of the method used has been made. The number of motor units and the peak terminal latency of the deep peroneal nerve did not differ significantly between the two groups, whilst the size of MUAP was significantly reduced in the patient group. Our findings are in accordance with the traditional concept of Duchenne muscular dystrophy as a primary myopathy, and cast doubts upon the evidence supporting the neurogenic theory of muscular dystrophy.
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