Abstract
In vitro experiments show that therapeutic doses of unfractionated and low-molecular-weight heparins inhibit fibrin polymerization. Sodium and calcium salts of unfractionated heparins exhibit antipolymerization activity. The activity of low-molecular-weight heparins is higher, at 0.75 anti-Xa units/ml fraxiparin produces a stronger inhibiting effect on polymerization of fibrin monomers than clivarin.
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