Abstract
In rats with complete spinal cord transection, one group received alpha methyl tyrosine to block norepinephrine production, one group received dibenamine to block the effect of excess norepinephrine on the blood vessels, and one group served as a control group. None of the animals showed any evidence of long motor tract regeneration. The amount of cystic change in the rostral and caudal ends of the transected cord was unaffected by either treatment. The reported decrease in hemorrhagic necrosis that occurs when production of norepinephrine is inhibitled in animals with blunt cord trauma could not be reproduced in animals with complete cord transection. Attempts to block the effect of excess norepinephrine on blood vessels with dibenamine failed to inhibit local cord necrosis in the rats.
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