Abstract
The ovarian hormones, estradiol and progesterone, are believed to act at specific sites in the female brain to induce sexual receptivity. Septal lesions which have been comprehensively studied in the rat, were carried out on female hamsters. Lordosis behavior was quantified as an index of female sexual receptivity. Septal lesions were found to cause a significant increase in female sexual receptivity in hamsters primed with 3 or 9 μg estradiol benzoate (EB) and 200 μg progesterone (P), but low levels of lordosis were not enhanced in septal lesioned female hamsters primed with 1 μg EB + 200 μg P. The facilitation of lordosis seen in septal lesioned hamsters is similar to that observed in female rats. However, our results do not confirm increases in sensitivity to estradiol in the hamster.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
