Abstract
Juvenile turbot (Scophthalmus maximus) were exposed to acaricide fenpyroximate, and the subsequent histological, enzyme activity changes of liver tissue were examined. Histopathological examinations showed that the turbot liver is the target organ for the acaricide fenpyroximate. Analysis of liver antioxidant enzymes, superoxide dismutase (SOD), catalase (CAT) and glutathione peroxidase (GPX), in the exposed turbots also showed similar changes. These changes indicated that fenpyroximate caused the oxidative stress on the turbot liver, which induced the damage of antioxidant enzymes system and possible interruption of the mitochondrial redox respiratory chain. This could be one toxic mechanism of fenpyroximate to the juvenile turbot.
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