Abstract
Several plant lectins markedly reduce the numbers of virus plaque-forming units released by cultures of different cell types, treated either before or after infection. Antiviral activity, which is not due to activation of the interferon system, is long lived and is demonstrable against a variety of enveloped and nonenveloped cytocidal RNA and DNA viruses. Although several mechanisms are responsible for the total antiviral effect, depending upon the virus studied, major effects seem to be the blocking of virus release, agglutination of cell-associated as well as liberated virus, and, possibly, diminished production of virus.
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