Abstract
Nitric oxide (NO) has been ascertained to play an important role in signal transduction in the central nervous system, functioning as a short-lived modulator and/or second messenger1,32. There is a functional link between NO and glutamatergic neurotransmission. NO is involved in synaptic plasticity, for example, in long-term potentiation, memory and learning, in the hippocampus4, which phenomena represent physiological effects of glutamate as a transmitter. Glutamate also participates in the development of ischemic brain injury, and therefore, NO is likely to be associated with mechanisms of neuronal damage4.
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