Abstract
Patients with active acromegaly often develop diabetes mellitus (DM) or impaired glucose tolerance (IGT). Treatment with somatostatin analogues (SA) does not only inhibit GH secretion but may also impair insulin secretion. In order to evaluate the influence of SA on glucose metabolism we investigated insulin resistance (IR) and β-cell function (HOMA-β), using the recommended combination of HOMA-R and HOMA-β model.
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