Abstract
Intracellular soluble microtubule protein levels were determined in cultured mammalian cells following X-irradiation. Microtubule protein levels rose along with corresponding RNA and total soluble protein levels as cells accumulated in G 2 and declined as cells resumed division. These results suggest that radiation-induced division delay of mammalian cells does not arise from a radiation-induced defect in synthesis and accumulation of microtubule protein.
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