Abstract
We describe a series of kinetic Monte Carlo simulations of post-cascade radiation damage evolution in α-iron that illustrates the part played by elastic interaction between defects. Elastic interactions are included as a bias to the diffusion of mobile point defects and defect clusters. The simulations show that recombination fractions are reduced, and vacancy clustering is enhanced. The sensitivity of these effects to temperature, cascade energy, and geometric description of vacancy clusters is also investigated.
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