Abstract
Dextran in doses of 1 to 1.5 g/ day inhibited production of cortisone hyperlipemia in rabbits and partially reversed the hyperlipemia produced by cortisone. Alterations in fat transport, fat mobilization or in clearing factor action are considered as the basis for antagonism between dextran and hyperlipemic action of cortisone.
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Schedule a callMore From: Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine (New York, N.Y.)
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