Abstract
and pepsin secretions were measured by titration and the Anson method, respectively. Stages of the measurements were basal (first and second), stimulated and returnedbasal. Results: Basal acid and pepsin secretions in the paraoxon group (14.61± 1.46 and 2.97± 0.32 g/15 min, respectively) were more than those in the control group (7.88± 0.26 and 0.55± 0.06 mol/15 min). Although pentagastrinstimulated acid secretion in all the three groups was more than basal acid secretion, there were no differences between the groups. Stimulated pepsin secretion in the paraoxon group was significantly more than that in the control group. Conclusion: Paraoxon, in chronic exposure, increases gastric acid and pepsin secretion.
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