Abstract
Stressor-induced decreases in glutathione (GSH) concentrations were demonstrated in the liver and the gastric mucosa. Either food deprivation and/or the time of day the stressor was administered could affect the extent of stressor-induced decreases in GSH. The system described may be useful for further exploration of potential stressor-induced alterations in susceptibility to chemical carcinogens and/or other toxins whose expression of biological activity may be dependent upon GSH.
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