Abstract
Atmospheric ammonia (NH/sub 3/) is produced by a number of industrial sources however it is the biological action of urease-containing bacteria upon urine and fecal waste products that is responsible for producing NH/sub 3/ in laboratory animal facilities. Since NH/sub 3/ is a primary irritant, studies emphasizing the pulmonary toxicity of both acute and repeated exposures have appeared. Since the mouse has proven to be a sensitive animal model for use in NH/sub 3/ toxicity studies and work in the rat has produced only minimal microsomal changes, the present study was undertaken to determine whether acute or repeated exposure to NH/sub 3/ gas inhibits selected microsomal enzyme activity in the mouse.
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