Abstract
Intraventricular administration of 25μg/kg of adrenaline induced consistently a delayed rise in gastric pepsin output in the Heidenhain pouch dog, wich is referable due to the release of ACTH. This response was blocked by pituitary stalk section and also significantly suppressed in pepsin output by electrolytic lesions placed in the median eminence and posterior hypothalamic regions.Ten μg/kg of adrenaline injected into the median eminence and posterior hypothalamic regions. caused a significant increase in gastric pepsin secretion, while the same dose of adrenaline injected into the anterior hypothalamic regions failed to do. An intrahypophysial injection of 25μg/kg of adrenaline, either into the anterior or the posterior lobe, proved ineffective.An intracarotid injection of 100μg/kg of adrenaline had no stimulatory effects on gastric secretion.
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