Abstract
Background and Aim: Liver disease leads to quantitative as well as qualitative reduction in albumin concentrations. Reversible and irreversible oxidized forms of albumin increase with severity of cirrhosis, compromising with the binding and detoxifying functions and leading to decrease in effective albumin concentration(eAlb). Albumin isoforms were studied in cirrhotic children and compared with non-cirrhotic controls and those with decompensation.
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