Abstract
Chronic experiments on fistulized dogs showed that the pancreatic secretion stimulated by intraduodenal administration of acidified hydrolysine was depressed after bilateral supradiaphragmatic truncal vagotomy, and that the inhibitory influence of exogenous pancreatic enzymes on pancreatic secretion disappeared on postoperative days 10–16. Subsequently, the feedback inhibition of pancreatic secretion was slowly restored, but even 45 days after vagotomy it was still weaker than in intact dogs. It is concluded that the feedback inhibition of pancreatic secretion is centrally controlled from the duodenum via the vagus nerves.
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