Abstract
INTRODUCTIONPREVIOUS studies have established that buquinolate1 at concentrations of 0.00825% and 0.011% in feeds effectively controls mild to severe Eimeria tenella infections in the chicken. Although Eckman et al. (1968) showed that micronization of buquinolate enhances the efficacy of the drug, little information is available to indicate the mechanism by which the drug exerts its coccidiostatic effect. Edgar and Flanagan (1967) reported that buquinolate acts to inhibit the early asexual development of the parasite, but also found that some of the parasites were able to complete their developmental cycle in the ceca of buquinolate medicated birds. Thus, chickens fed this coccidiostat may show no clinical signs of E. tenella infection and still pass small numbers of oocysts, a phenomenon evidenced by the fact that the use of buquinolate does not interfere with the host’s development of active immunity to subsequent infection.The present study was undertaken to determine the…
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