Abstract
Celiac disease (CeD) is a long-term inflammatory disorder of the small intestine, triggered by the ingestion of gluten in genetically susceptible individuals. When gluten is consumed, it leads to damage to the intestinal lining, resulting in poor absorption of nutrients. Although nutrient malabsorption is a significant risk for various complications associated with CeD, genetic, immune, and environmental factors also play key roles. The clinical presentation of CeD can vary widely, ranging from no symptoms to severe symptoms, reflecting its impact on multiple organ systems. Currently, the only evidence-based treatment is a gluten-free diet (GFD), which can fully repair the intestinal damage and stop disease progression. If left undiagnosed, CeD can lead to serious complications in both children and adults.
Published Version
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