Abstract
PurposeDuring orthodontic treatment with fixed appliances, demineralization around brackets often occurs. The aim of this in vitro study was to investigate the effect of the caries-protective self-assembling peptide P11‑4 (SAP P11-4) on the shear bond strength of metal brackets.MethodsIn all, 45 extracted human wisdom teeth were available for the study. The teeth were randomly divided into 3 groups (each n = 15) and pretreated as follows: test group 1: application of SAP P11‑4 (Curodont Repair, Windisch, Switzerland) and storage for 24 h in artificial saliva; test group 2: application of SAP P11‑4; control group: no pretreatment with SAP P11‑4. A conventional metal maxillary incisor bracket (Discovery, Dentaurum, Ispringen) was adhesively bonded to each buccal surface. The shear bond strength was tested according to DIN 13990. After shearing, the Adhesive Remnant Index (ARI) was determined microscopically (10 × magnification). Analysis of variance (ANOVA) was used to check the groups for significant differences (α = 0.05). The distribution of the ARI scores was determined with the χ 2 test.ResultsThere was no significant difference in shear forces between the groups (p = 0.121): test group 1 = 17.0 ± 4.51 MPa, test group 2 = 14.01 ± 2.51 MPa, control group 15.54 ± 4.34 MPa. The distribution of the ARI scores between the groups did not vary (p-values = 0.052–0.819).ConclusionThe application of the caries protective SAP P11‑4 before bonding of brackets did not affect the shear bond strength. Therefore, pretreatment of the enamel surface with SAP P11‑4 shortly before bracket insertion can be considered.
Highlights
Dental caries is a biofilm-mediated, multifactorial, noncommunicable disease resulting in net mineral loss of dental hard tissues and is determined by biological, behavioral, psychosocial, and environmental factors [30, 42]
The incidence of new carious lesions that formed during orthodontic treatment was found to be 45.8% [47] and development of cavitations after treatment with multibracket appliances was reported in 26.9% of patients [17]
The present study aimed to investigate the influence of self-assembling peptide (SAP) P11-4 on shear bond strength of metal brackets to the enamel surface
Summary
Dental caries is a biofilm-mediated, multifactorial, noncommunicable disease resulting in net mineral loss of dental hard tissues and is determined by biological, behavioral, psychosocial, and environmental factors [30, 42]. Orthodontic treatments, especially with fixed appliances, provide a hurdle to oral hygiene and lead to patients becoming high-risk caries patients. Plaque that accumulates around the brackets is not removed to the same level as would be required, resulting in demineralization. It is reported that white spot lesions have a limited ability to improve after appliance removal and can be detectable many years after treatment [46]. The incidence of new carious lesions that formed during orthodontic treatment was found to be 45.8% [47] and development of cavitations after treatment with multibracket appliances was reported in 26.9% of patients [17]. It is even stated that the high treatment demand and occurrence of biofilm-related complications make orthodontic treatment a potential public health threat [44]
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