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Abstract
1. 1. Tritium release evoked by field electrical stimulation (FES) or high potassium (K +) from cat cerebral and femoral arteries prelabelled with 3H-noradrenaline was investigated. 2. 2. The release induced by FES and K + was reduced by Ca 2+ suppression and tetrodotoxin (TTX) but not by verapamil in both vessels. 3. 3. Tetraethylammonium (TEA) increased tritium release evoked by FES and K +, when TTX plus TEA were added together radioactivity secretion induced by FES was practically abolished. 4. 4. These results indicate that FES or K + induce exocytotic noradrenaline release mainly by propagated action potentials and by similar mechanisms in cerebral and femoral arteries.
Published Version
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