Abstract
Experiments on albino rats revealed that the skeletal changes characteristic of bone lathyrism (multiple exostoses, hypertrophy of junction cartilage plates, slipped epiphyses, “basophilic bone globules”), which are readily produced in normal rats by treatment with aminoacetonitrile (AAN), are virtually abolished in the totally paralyzed hind limbs of animals whose spinal cord has been transected between the tenth and the eleventh thoracic vertebrae. On the other hand, the periosteal bone hemorrhages, which are occasionally produced by AAN in intact animals, tend to occur even more readily in the paralyzed limbs and may occasionally be the site of ectopic cartilage formation.
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