Abstract
The writers showed previously that mice of the inbred line I/St are highly susceptible to infection with tuberculosis, whereas those of inbred line A2G are resistant [2]. Parameters of immunity to tuberculosis, such as the level of the delayed-type hypersensitivity (DTH) reaction and of antibody synthesis to tuberculin, were opposite in mice of these lines at the same times after infection: a high level of DTH and a low level of antibodies in A2G mice and the opposite picture in I/St mice [i]. These data confirm the earlier concept [3] of immunological causes of sensitivity and resistance to tuberculosis and the protective role of cellular immunity in this disease.
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