Abstract
RATIONALE: Lipopolysaccharide (LPS) leads to neutrophil recruitment to the lung and is associated with exacerbation of allergic airway inflammation and remodeling in humans and in animal models. We examined whether inhaled LPS could cause hyperinnervation of sensory C-fibers in the rat airway, likely resulting in enhancement of neurogenic airway inflammation. In addition, the preventive effect of procaterol, a β2-adrenoceptor agonist, on the increased neural mechanism after LPS inhalation was investigated.
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