Abstract
This study evaluated the effect of desensitizer and rewetting agent on dentin shear bond strength. One hundred thirty five bovine incisive teeth had their buccal surfaces ground down to produce a fl at superficial dentin surfaces and received the following treatments: G1- One Step Plus (OSP) and resin cylinder adhesive fi xation (RI); G2- Gluma One Bond (GOB) and RI; G3- Single Bond (SB) and RI; G4- Aqua-Prep (AP) + OSP and RI; G5- Gluma Desensitizer (GD) + GOB and RI; G6- GD + SB and RI; G7- GD + OSP and RI; G8- AP + GOB and RI; G9- AP + SB and RI. The specimens were stored at 37°C and 100% humidity for 24 hours and a shear bond test was performed with a mechanical testing machine, at a crosshead speed of 0.5 mm⁄min. The data were submitted to one-way ANOVA followed by the Tukey test (p<0.05). The results (MPa) were: G1: 10.75(2.64)a; G2: 10.28(2.58)a; G3: 11.63(4.59)a; G4: 10.93(4.88)a; G5: 10.15(3.95)a; G6: 11.82(4.14)a; G7: 9.85(2.15)a; G8: 5.48(1.94)b; G9: 10.62(2.83)a. The resistance of the GOB adhesive system was negatively affected by AP application. The use of desensitizer and rewetting agent does not compromise the bond strength when they are compatible with the adhesive system used.
Highlights
The hydrodynamic theory of dentin sensitivity is widely accepted
No statistically significant difference was found among the shear bond strength values of bonding systems applied alone and when they were associated with Gluma Desensitizer
The homopolar methacrylate group of the HEMA molecule has a high affinity for hydrophobic monomers1
Summary
The hydrodynamic theory of dentin sensitivity is widely accepted. Dentin hypersensitivity is thought to be caused by displacement of dentin fluid within the dentin tubules. The dentin tubules of hypersensitive teeth are open, more numerous and larger than in normal teeth. Dentin sensitivity presents a challenge to the dentist. Modern treatments for hypersensitive teeth are intended either to reduce tubular fluid movements by reducing dentin permeability or to reduce the excitability of intradental nerves with neurally active agents. Tubule occlusion is thought to reduce subjacent pulpal inflammation
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