Abstract
Body temperature in mice fed with a low-potassium diet decreased in a parallel way to the diminution of plasma potassium concentration. Both parameters were restored after potassium replenishment. Hypokalemia produced by treatment with fludrocortisone, a synthetic mineralocorticoid, did not significantly decrease mouse body temperature. The hypothermia observed in the potassium deficient mice was prevented by treatment with fludrocortisone, in spite of the fact that plasma potassium levels were remarkably reduced. The transient hyperthermic response induced by stress in control animals was also present in the potassium deficient mice. Dexamethasone prevented stress-induced hyperthermia both in control and potassium-deficient animals. The results may be partially explained by the reported effects of potassium and fludrocortisone on Na/K ATPase.
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