Abstract
SummaryIntracellular sodium concentration of stimulated rat gastrocnemius muscle increased 3-fold while potassium decreased by almost one-third after 20 minutes of stimulation (20/sec). Dibenzylene abolished these changes and strikingly reduced the amplitude of muscular contraction. Pitressin (80 mU intravenously) in Dibenzylene treated rats restored both the cationic shifts and muscular contractions during its period of effectiveness. We suggest that the ability of Pitressin to restore work performance is brought about by a direct action on cationic exchanges across the muscle membrane.
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
