Abstract
Nitric oxide (NO) is an intracellular messenger molecule involved in modulating cell proliferation and apoptosis in a variety of cells. The present study was designed to determine if NO modulated the proliferation of human lung fibroblasts. Human fetal lung fibroblasts (HFL-1) were maintained in cell culture in Dulbecco's Modified Eagle's Medium in the presence of 4% fetal calf serum. Cell proliferation was determined by enumerating cells with a Coulter counter. Inhibition of endogenous NO production by N-nitro-L-arginine methylester had no effect on HFL-1 cell proliferation.
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